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GHK is a high-affinity copper-binding peptide that has been proposed to function as a physiological copper carrier and delivery agent moving copper between proteins and binding sites in plasma and in tissues, with downstream consequences for processes that depend on copper availability and on GHK-dependent signaling

It has been shown to influence the expression of genes involved in angiogenesis (e.g., egr-1), stabilize endothelial integrity, and facilitate the recruitment of fibroblasts to injury sites 4

BPC-157's primary mechanisms (VEGF/angiogenesis and gut-lining signalling) are the most extensively researched and best-characterised in the published literature

There is no need to reduce the particle size of freely water soluble drugs, e.g

Section 5 says